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cetuximab (Lupitux / Cetuxa / ENZ124)

✓ Approved

Lupin Limited · EGFR · 单克隆抗体

什么是 cetuximab?

cetuximab 是一种单克隆抗体,由Lupin Limited研发。该药已获批,用于治疗相关适应症,给药途径:Injectable (Others)、Intravenous (IV)。

药物档案

商品名Lupitux, Cetuxa, ENZ124
公司Lupin Limited
药物类别单克隆抗体, 抗体
分子靶点EGFR
给药途径Injectable (Others), Intravenous (IV)
状态Approved

作用机制

分子靶点

cetuximab 作用于 1 个分子靶点:

EGFRepidermal growth factor receptor (ERBB1, NNCIS)
需要更深入的分析?Noah AI 可解释复杂机制并与同类药物比较。

治疗适应症

cetuximab 针对 1 个适应症,涉及 1 个治疗领域。

治疗领域疾病/病症分期
Neoplasms benign, malignant and unspecified (incl cysts and polyps)Head and neck cancer metastatic✓ Approved

相关研究文献

PubMedHealth services insights2026-08-02

Adverse Event Profiles of Monoclonal Antibodies: A Descriptive Analysis of FAERS Data.

Alem Ghada G, Ahmed Nehad Jaser NJ, Ahmed Maram M, Balaha Mohamed M et al.

Monoclonal antibodies (mAbs) are bioengineered molecules designed to replicate the immune system's precise targeting of pathogens and abnormal cells. While monoclonal antibodies (mAbs) are targeted therapies, they carry risks like immunogenic reactions and drug interactions. The current study utilizes FAERS to describe the distribution of reported adverse events, patient demographics, and reporting patterns associated with selected monoclonal antibody therapies. The analysis included all monoclonal antibody adverse event reports in FAERS through March 31, 2025, with subsequent reports and non-monoclonal antibody drugs excluded, and utilized descriptive statistics to present results as numbers and percentages. FAERS data through March 31, 2025, for 18 monoclonal antibodies showed distinct adverse event (AE) patterns. Most AEs occurred in adults aged 18-64, with females reporting more events except for cancer-related mAbs such as cetuximab (67.09% males) and ipilimumab (64.15% males). Healthcare professionals were the primary reporters, though some drugs-like adalimumab (72.13%)-had mainly consumer reports. Common AEs included injection-site pain (7.44%), rash, in addition to reports categorized as "drug ineffective," a MedDRA preferred term used in FAERS to reflect reporter perception of treatment failure (e.g., 17.96% for secukinumab; 28.14% for tocilizumab). The most frequently reported events included treatment-ineffectiveness perceptions and administration issues, which warrant further investigation in controlled studies. Some reports included product use issues such as dose omission or incorrect administration. However, due to the spontaneous reporting nature of FAERS, the relationship between adherence-related issues and clinical outcomes cannot be definitively established. The findings of the study describe reporting patterns in FAERS and may serve as a basis for future pharmacovigilance or epidemiologic research.

PMID 42540027
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PubMedCancer medicine2026-07-31

The Impact of the G8 Score on Survival Outcomes in Patients With Head and Neck Cancer Undergoing Concurrent Radiotherapy With Cetuximab due to Contraindications With Cisplatin.

Kiraz Esin E, Gündoğ Mete M, Gönül Rıdvan R, Kara İrfan İ et al.

Patients unable to receive cisplatin due to age, comorbidities, or reduced performance status are commonly treated with concurrent radiotherapy (RT) and cetuximab. This study evaluated the prognostic significance of the Geriatric 8 (G8) score and the Charlson Comorbidity Index (CCI) in predicting survival outcomes in this population. We retrospectively analyzed data from 52 patients with locally advanced or loco-regional recurrent head and neck squamous cell carcinoma (HNSCC) who were treated with RT and cetuximab between 2011 and 2024. Patients with p16-positive oropharyngeal or nasopharyngeal cancers were excluded. Overall survival (OS) and cancer-specific survival (CSS) were assessed according to G8 and CCI scores using ROC analysis, Kaplan-Meier curves, and Cox regression. The median follow-up was 50 months. A G8 cut-off of 9.75 was identified (AUC: 0.713, p < 0.01). Patients with G8 ≥ 9.75 showed significantly improved OS (median: 47 vs. 7 months, p < 0.01) and 4-year CSS (66% vs. 32%, p < 0.01). In the definitive treatment group, 4-year CSS was 68% in high-G8 patients vs. 42% in low-G8 patients. In the recurrence group, 4-year CSS was 63% among high-G8 patients, whereas no patient with a low G8 score survived beyond 4 years. Multivariate analysis identified G8 < 9.75 (HR: 4.72, p < 0.01) and RT dose < 60 Gy (HR: 3.01, p = 0.01) as independent predictors of poor OS. No significant association was observed between CCI and survival. The G8 score is a valuable prognostic tool for cisplatin-ineligible HNSCC patients treated with RT and cetuximab. Low G8 scores were strongly associated with worse outcomes, highlighting the need for alternative therapeutic strategies in this high-risk subgroup.

PMID 42532979
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PubMedThe Laryngoscope2026-07-30

Treatment of Locally Advanced Layngohypopharyngeal Cancers: A Network Meta-Analysis.

Capriotti Vincenzo V, Cin Elisa Dal ED, De Stefani Agostina A, Nardone Massimiliano M et al.

To compare the efficacy of non-surgical organ-preservation strategies for locally advanced or resectable laryngeal and hypopharyngeal squamous cell carcinoma (SCC). Randomized controlled trials (RCTs) published through June 30, 2025, identified via systematic database and trial-registry searches and reference screening. We performed a systematic review and Bayesian random-effects network meta-analysis of RCTs enrolling adults with locally advanced/resectable laryngeal/hypopharyngeal SCC. Treatments included surgery plus postoperative radiotherapy (RT), RT alone, concurrent cisplatin-based chemoradiotherapy (CTRT), induction chemotherapy followed by RT/CTRT, altered-fractionation RT, dose-modified CTRT, and RT ± cetuximab. Effect estimates were synthesized as hazard ratios (HRs) and odds ratios (ORs). Treatments were ranked using surface under the cumulative ranking curve (SUCRA). Fifteen RCTs (n = 4395; 13 strategies) were included. For laryngeal preservation, cisplatin-based CTRT ranked highest (SUCRA 0.73), followed by induction TPF → RT (0.72) and RT + cetuximab (0.61). All non-surgical strategies outperformed surgery plus postoperative RT for laryngeal preservation (HR 0.34; 95% CI, 0.20-0.58). For overall survival, induction PF → RT showed the highest probability of benefit (SUCRA 0.68), with overlapping credible intervals across leading regimens. For locoregional control, induction TPF → RT ranked best (SUCRA 0.94). Sensitivity analyses supported robustness. Concurrent cisplatin-based CTRT remains the most consistent organ-preservation strategy. Induction PF → RT may offer modest overall survival benefit, whereas sequential TPF → RT appears superior for locoregional control. Treatment should be individualized by comorbidity, functional goals, and institutional expertise; head-to-head trials and incorporation of immunotherapy/targeted agents are needed. N/A.

PMID 42528018
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PubMedAsian Pacific journal of cancer prevention : APJCP2026-07-29

Oral Squamous Cell Carcinoma: A New Era in Molecular Mechanisms and Emerging Targeted Therapies.

Harendra Bhavana B, P Ashwini A, C P Kavana K, Nagaraj Abhigna A et al.

Oral squamous cell carcinoma (OSCC), the most common oral cancer, presents a clinical challenge due to its complex tumor microenvironment (TME), dysregulated pathways, and poor prognosis. Current methods of diagnosing OSCC use liquid biopsy technologies (ctDNA/microRNAs/exosomes) instead of relying solely on traditional methods such as open surgical biopsy. Liquid biopsy technologies provide non-invasive ways to detect and monitor OSCC in early stages, compared with traditional open surgical biopsy methods. Treatment of OSCC currently relies on chemotherapeutics (cisplatin/5-FU), radiotherapy, and targeted agents (cetuximab). However, resistance is acquired due to TME remodelling (tumor microenvironment) and/or due to epithelial-mesenchymal transition (EMT) through processes such as ABC transporter efflux. This review elucidates key molecular mechanisms, including PD-L1-mediated immune evasion, PI3K/AKT/mTOR hyperactivation, EGFR overexpression, and NF-κB-driven inflammation, which promote proliferation, metastasis, and therapy resistance. One area of study involves the use of nanotechnology to convert phytocompounds (medicinal plants) into therapeutic agents by embedding phytocompounds into nanoparticles created using green synthesis techniques. EPR (Enhanced Permeability and Retention), ligand functionalization for OSCC targeting, improved bioavailability, and reduced toxicity are all advantages that the aforementioned systems provide, offering an opportunity to synergistically develop new therapies with chemotherapeutics for overcoming resistance. While numerous preclinical studies demonstrate that these newly developed therapies show increased efficacy compared with current therapy options, further work is needed in areas such as standardization, scaling, and clinical translation. As such, the importance of developing pathway-informed diagnostic tests and developing therapies that exploit pathway-specific activity with phytocompounds is emphasized. In addition, large-scale studies should be considered to evaluate the effectiveness of a pathway-informed approach in assessing and differentiating OSCC and personalized therapy strategies, to improve OSCC survival outcomes.

PMID 42522799
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PubMedOral oncology2026-07-28

Can the Reported Benefit of Cetuximab After Immune Checkpoint Inhibitor Failure Be Attributed to Cetuximab Alone?

Bagchi Pareeksit Rajkumar PR, Yennemadi Swathi S

PMID 42508376
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PubMedPharmaceuticals (Basel, Switzerland)2026-07-28

Overcoming Resistance to Anti-EGFR Therapies: Mechanisms of Cetuximab and Panitumumab Resistance and Emerging Combination Strategies.

Henrykowska Gabriela G, Bartusik-Aebisher Dorota D, Dynarowicz Klaudia K, Ramesh Tamil Selvan TS et al.

Cetuximab and panitumumab are anti-EGFR monoclonal antibodies widely used for the treatment of colorectal cancers. However, due to various mechanisms of resistance to these targeted therapies, the patients' responses vary. These resistances remain a major obstacle in treatment and overcoming them has become a key emphasis of current therapeutic strategies. Intrinsic and acquired resistance often lead to reactivation of downstream signaling pathways, mainly the RAS-RAF-MEK-ERK (MAPK pathway) and PI3K-AKT axes. Prior existing mutations in KRAS, NRAS, and BRAF result in primary resistance by constantly activating the signals, irrespective of EGFR inhibition. That said, acquired resistance manifests under therapeutic burden through the process of clonal evolution via KRAS and BRAF alterations, restoring MAPK pathway activity despite EGFR inhibition. In addition to those mutations, tumor cells exploit mechanisms independent of EGFR, such as the pathway bypass, which includes amplification of ERBB family receptors like HER2 (ERBB2) and activation of MET signaling. To overcome these resistances, novel strategies have emerged, which target multiple nodes within the oncogenic networks. Such methods include vertical pathway inhibition, multi-kinase inhibition, liquid-biopsy-guided therapy, and anti-EGFR rechallenge. Reactivation driven by secondary mutation can be prevented by targeting multiple nodes within the MAPK cascade simultaneously, which is referred to as the vertical pathway inhibition. Overall, this review underscores that overcoming therapeutic resistance requires a multidimensional approach that integrates molecular profiling, rational combination therapies, and adaptive treatment. Finally, these advances underscore the shift toward precision oncology, where therapy is tailored to tumor evolution, leading to improved response and patient outcome.

PMID 42515723
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