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piperacillin (piperacillin, KRKA / Isipen)

✓ Approved

Krka · 小分子 · 小分子

什么是 piperacillin?

piperacillin 是一种小分子,由Krka研发。该药已获批,用于治疗相关适应症,给药途径:Unknown。

药物档案

商品名piperacillin, KRKA, Isipen
公司Krka
药物类别小分子
给药途径Unknown
状态Approved

治疗适应症

piperacillin 针对 1 个适应症,涉及 1 个治疗领域。

治疗领域疾病/病症分期
Infections and infestationsSalmonellosis✓ Approved

相关研究文献

PubMedPediatric critical care medicine : a journal of the Society of Critical Care Medicine and the World Federation of Pediatric Intensive and Critical Care Societies2026-08-06

Piperacillin Population Pharmacokinetics in Children Supported With Extracorporeal Membrane Oxygenation.

Arsène Adrien A, Oualha Mehdi M, Rambaud Jérôme J, Benaboud Sihem S et al.

To characterize piperacillin population pharmacokinetics (PK) in critically ill children with and without extracorporeal membrane oxygenation (ECMO) support and to optimize piperacillin dosing regimens to attain the PK target of 100% free time above minimum inhibitory concentration (fT > minimum inhibitory concentration). Post hoc secondary analysis of a two-center, population PK modeling and simulation study (registered at clinicaltrials.gov, NCT02539407). PICUs at tertiary care centers, September 2015-November 2021. Thirty-three children supported with ECMO and 50 non-ECMO control patients treated with piperacillin (± tazobactam). Administration of piperacillin at 300 mg/kg/d by continuous infusion or 75 mg/kg every 6 hours by intermittent infusion. A one-compartment linear-elimination model described best the piperacillin data. Clearance was driven by body weight and estimated glomerular filtration rate (eGFR). In a typical 11-kg patient, ECMO circuit volume-to-weight ratio increased the volume of distribution by 34%. Monte Carlo simulations across eGFR strata (30-60, 60-90, 90-150, 150-200, 200-300 mL/min/1.73 m2) showed that a continuous infusion of 300 mg/kg/d allowed greater than or equal to 90% of target attainment in all groups. Patients with an eGFR greater than 200 mL/min/1.73 m2 required 400 mg/kg/d to have 90% of target attainment. Continuous infusion of 300 mg/kg/d piperacillin reliably achieves PK target across all renal-function ranges in children undergoing support with ECMO. Dose escalation to 400 mg/kg/d is required for augmented renal clearance (eGFR > 200 mL/min/1.73 m2).

PMID 42560139
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PubMedFrontiers in medicine2026-08-06

Clonal patient-to-patient spread is associated with multidrug-resistant Acinetobacter baumannii infections in a Chinese pediatric hospital: an integrated phenotype-genotype-PFGE surveillance study.

Luo Wanjun W, Gong Xuyang X, Liu Sijing S, Deng Shiyong S et al.

Carbapenem-resistant Acinetobacter baumannii is a critical-priority pathogen in pediatric hospitals, but the relative roles of clonal transmission and environmental reservoirs remain unclear. We analyzed 42 non-duplicate A. baumannii isolates collected between 2020 and 2023 from clinical specimens (n = 28), medical waste (n = 8), and hospital-setting samples (n = 6) at Wuhan Children's Hospital. Antimicrobial susceptibility testing, PCR-based resistance gene detection, pulsed-field gel electrophoresis, and crystal-violet biofilm assays were performed. Clinical isolates showed high multidrug resistance (75.0%, 21/28), with 75.0% resistant to piperacillin-tazobactam, ceftazidime, imipenem, meropenem, and ciprofloxacin. All isolates remained susceptible to tigecycline. OXA-23 was detected in all carbapenem-resistant clinical isolates, and aac(6')-Ib-cr was present in all fluoroquinolone-resistant isolates. One dominant clinical clone, T11, accounted for 57.1% of clinical isolates and included one OXA-23/NDM co-producing isolate. PFGE revealed no clonal overlap between clinical and non-clinical isolates; all medical-waste and hospital-setting isolates were fully susceptible. Biofilm formation was detected in 97.6% of isolates; among clinical isolates, biofilm intensity was inversely associated with multidrug resistance, with susceptible isolates forming stronger biofilm (Fisher's exact p < 0.001). Pediatric multidrug-resistant A. baumannii infections in this hospital were most consistent with patient-to-patient dissemination of a dominant resistant clone rather than with contamination from the hospital-setting or medical-waste reservoirs sampled here, although the small environmental sample cannot definitively exclude such reservoirs. Phenotype-genotype-PFGE surveillance provides actionable guidance for infection control in resource-limited pediatric settings.

PMID 42559283
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PubMedFrontiers in veterinary science2026-08-05

Phenotypic antimicrobial susceptibility and genomic resistance determinants in canine otitis-associated Pseudomonas aeruginosa isolates.

Veres Mercédesz Adrienn MA, Tóth Zsófia Anna ZA, Illés Enikő E, Mag Patrik P et al.

Pseudomonas aeruginosa is a clinically important opportunistic pathogen in canine otitis externa, particularly in chronic, recurrent or treatment-refractory cases. Its low outer-membrane permeability, biofilm-forming capacity, efflux systems and ability to acquire additional resistance determinants complicate empirical therapy and make local surveillance essential. This study integrated broth microdilution-based minimum inhibitory concentration (MIC) testing with whole-genome analysis to characterize antimicrobial susceptibility patterns and genomic resistance determinants in canine otitis-associated P. aeruginosa isolates from Hungary. A total of 110 isolates were tested against 14 antimicrobial or antiseptic agents. Official clinical breakpoints and European Committee on Antimicrobial Susceptibility Testing (EUCAST) epidemiological cut-off values were applied where available, while agents lacking validated interpretive criteria were evaluated descriptively. A phenotypically selected subset of 70 isolates, chosen to cover the major MIC ranges, resistance profiles and modified MAR-index spectrum observed in the full collection, was subjected to long-read sequencing, and all 70 genome assemblies were included in comprehensive antibiotic resistance database (CARD)-based resistance gene screening, multi-locus sequence typing (MLST) and genome quality assessment. High frequencies of elevated MICs were detected for several clinically relevant agents. The highest proportions above interpretive thresholds were observed for enrofloxacin (109/110, 99.1%), marbofloxacin (99/110, 90.0%), piperacillin-tazobactam (75/110, 68.2%), ceftazidime (71/110, 64.5%) and tobramycin (62/110, 56.4%). The median modified multiple-antimicrobial-resistance index, calculated only from agents with interpretable resistance thresholds, was 0.56. Genomic analysis showed a broad intrinsic resistance background dominated by efflux-associated determinants and recurrent aminoglycoside, beta-lactam and fluoroquinolone-associated genes, including APH(3')-IIb, Mex-family efflux components, Opr-associated transporters, arnA, basS, PDC/OXA beta-lactamases and crpP in a subset of isolates. These findings support the need for culture- and MIC-guided therapy in canine P. aeruginosa otitis and support the value of including companion-animal clinical isolates in national and regional antimicrobial resistance surveillance frameworks.

PMID 42553024
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PubMedFrontiers in microbiology2026-08-05

Antimicrobial susceptibility and virulence traits of Escherichia coli in healthy South American camelids in Germany.

Moe Aung Zaw AZ, Heydel Carsten C, Wagner Henrik H, Ulrich Lisa L et al.

South American camelids (SACs) are gaining popularity in the leisure sector, leading to human contact. However, data on pathogenic, zoonotic, and antimicrobial-resistant (AMR) bacteria in SACs remain limited. This study investigated the occurrence of AMR and virulence determinants in Escherichia coli isolated from feces of healthy alpacas and llamas in Germany. Between April 2021 and February 2023, longitudinal sampling was conducted on up to 20 animals per farm from 10 farms across six German federal states, with four sampling rounds at 6-month intervals. One random E. coli isolate per sample was obtained using a non-selective approach and tested for susceptibility to 14 antibiotics by broth microdilution, following EUCAST guidelines. In parallel, selective screening was performed to detect colistin- and carbapenem-resistant strains as well as ESBL/AmpC β-lactamase producers. Whole-genome sequence data from non-selectively obtained isolates mainly from round A and selectively obtained isolates from all rounds were used to identify AMR determinants and virulence-associated markers. Overall, 706 non-selectively isolated E. coli showed low resistance rates: sulfamethoxazole (4.5%), trimethoprim (1.8%), tetracycline (1.6%), ampicillin (1.4%), enrofloxacin (0.4%), cefotaxime (0.4%), colistin (0.1%), amoxicillin/clavulanate (0.1%), and piperacillin/tazobactam (0.1%). Eleven isolates exhibited multidrug-non-wild-type (MD-non-WT) phenotypes to ≥3 antimicrobial classes. Selective screening identified 18 mcr-1 and 70 cefotaxime non-WT isolates, but no carbapenem non-WT isolates. Resistance genes included bla CTX-M-1/14/15/27/55 (n = 54), bla DHA-1 (n = 4), bla CMY-2 (n = 4) and ampC promoter mutations (C42T) (n = 2). All colistin-resistant isolates carried mcr-1.1 on an IncI2 plasmid and originated from a single farm. Multilocus VNTR analysis revealed high genetic diversity with farm-specific clustering and temporal shifts. Although overall AMR prevalence was low, localized presence of ESBL- and AmpC-β-lactamase-producing and colistin-resistant E. coli indicates that SACs may represent AMR sources. Virulence profiling revealed few pathogenic E. coli except Shiga toxin-producing E. coli. These included low-virulence stx subtypes (stx1c, stx2b, and stx2f) and the high-virulence subtype stx2d, associated with enterohemorrhagic E. coli (EHEC) causing hemorrhagic colitis and hemolytic uremic syndrome in humans. Notably, detection of an EHEC isolate belonging to the O63:H6/ST583 lineage, a serotype/sequence type previously associated with severe clinical outcomes, highlights a zoonotic risk associated with SACs.

PMID 42553388
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PubMedThe Journal of antimicrobial chemotherapy2026-08-04

Impact of modified microbiology reporting on the treatment of low-risk AmpC Enterobacterales infections: a multi-centre controlled interrupted time series study.

Granger Marie-Félixe MF, Elligsen Marion M, McCready Janine J, Li Xena X XX et al.

We assessed the impact of a modified microbiology report and cascade that selectively reported narrower-spectrum β-lactams for low-risk AmpC Enterobacterales (AmpC-E). We conducted a controlled interrupted time series analysis to assess the proportion of patients receiving ceftriaxone or piperacillin/tazobactam on the fourth day of antibiotic treatment for low-risk AmpC-E infection (Serratia marcescens, Morganella morganii, Providencia spp., Citrobacter koseri) compared with a high-risk AmpC-E (Enterobacter cloacae complex, Klebsiella aerogenes, Citrobacter freundii) control group. Secondary outcomes included a pre-post measurement of antibiotic use on the fourth day of therapy, death at 30 days, and recurrent infection at 90 days. Patients with low-risk AmpC-E infections were more likely to receive ceftriaxone or piperacillin/tazobactam compared with high-risk AmpC-E controls [incidence rate ratio (IRR) 39.8, 95% CI 1.8-871.7, P = 0.02]. There was an upward shift and trend in ceftriaxone and piperacillin/tazobactam use during the implementation period in the low-risk AmpC-E group compared with the control group (IRR 3.3, 95% CI 0.9-11.9, P = 0.07 and slope 1.3, 95% CI 1.0-1.8, P = 0.10), with an associated reduction in fluoroquinolone use. During the implementation period, 30 day mortality (13.8% versus 12.1%, P = 0.70) and recurrent infection at 90 days (6.4% versus 5.7%, P = 0.83) were similar between low-risk and high-risk AmpC-E groups. Modification of microbiology reporting for low-risk AmpC Enterobacterales infections to selectively report narrower-spectrum β-lactams resulted in a rapid and sustained increase in use of ceftriaxone and piperacillin/tazobactam across two hospitals. Aligning this microbiology reporting with guidelines would have scalable impact on hospital antimicrobial stewardship.

PMID 42550165
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PubMedThe Pediatric infectious disease journal2026-08-04

Nonfermenting Gram-Negative Bacilli Other Than Pseudomonas and Acinetobacter Species in Pediatric Blood Cultures: A Multicenter European Analysis.

Bianco Gabriele G, Gryndlerova Anezka A, Schønning Kristian K, van Kleef-van Koeveringe Stefanie S et al.

Nonfermenting Gram-negative bacilli other than Pseudomonas and Acinetobacter species are increasingly recognized as opportunistic pathogens in pediatric populations, yet their epidemiology and antimicrobial resistance remain poorly defined. We conducted a multicenter retrospective analysis across 56 hospital centers in 25 European countries, including pediatric blood culture isolates collected between January 2020 and December 2024. Species distribution and antimicrobial susceptibility profiles according to European Committee on Antimicrobial Susceptibility Testing (EUCAST) and Clinical and Laboratory Standards Institute (CLSI) current guidelines were analyzed. A total of 382 isolates were included, spanning 19 genera and 40 species. Stenotrophomonas maltophilia was the most frequently identified species (46.9%; n = 179), followed by Roseomonas mucosa (10%; n = 38) and Sphingomonas paucimobilis (7.6%; n = 29). Susceptibility profiles were heterogeneous, with notable resistance across several species. For S. maltophilia, resistance to trimethoprim/sulfamethoxazole exceeded 10% (EUCAST), while full in vitro susceptibility to aztreonam/avibactam was observed. Preserved activity of carbapenems but high resistance to ceftazidime (EUCAST/CLSI), piperacillin/tazobactam (EUCAST/CLSI) and ciprofloxacin (CLSI) was observed for S. paucimobilis. Achromobacter several species showed concerning resistance patterns to carbapenems, piperacillin/tazobactam and trimethoprim/sulfamethoxazole (CLSI). Nonfermenting Gram-negative bacilli other than Pseudomonas and Acinetobacter species were frequently associated with healthcare exposure and complex resistance profiles. Their management is challenged by a lack of evidence and standardized susceptibility criteria. To optimize management, enhanced surveillance, improved diagnostic standardization and further clinical studies on emerging therapies are needed.

PMID 42547924
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